penis elargement photo

Erectile dysfunction (ED) or impotence can be a pointer to many underlying serious diseases. ED usually refers to the repeated inability to get or maintain an erection while performing the sexual act. Recently conducted studies have shown that 80 per cent of erectile dysfunction (ED) cases are related to the physical vascular system and only 20 per cent to psychological problems. Though ED is very common but its impact on our life can be devastating. But in spite of the risks and dangers associated with it, men tend to ignore ED and prefer to suffer in silence. They often experience a drop in self- esteem and believe they will become the subject of jokes if they seek treatment for ED or any sexual ailment. But the real fact is that we need to take erectile dysfunction as seriously as coronary artery disease, hypertension, diabetes, high cholesterol, stress, smoking, obesity and lack of exercise. Never ignore ED as it’s not just a sex-related problem. In most of the circumstances, ED may be the first symptom to develop a heart disease or stroke may follow. One clear connection between ED and the risk of heart disease or stroke has to do with changes developing in the small blood vessels of the man's penis that initiate ED. Similar changes in the larger blood vessels such as those leading to the brain or heart can eventually result in heart attack or stroke. So those men who are experiencing erection problems should not hesitate to seek immediate medical help. ED can be treated effectively, and in case of presence of other hidden ailments, these can be detected and treated early. The treatment of ED is not a big issue as wide range of drugs are available to cater to this need. Apart from the available branded drugs you can also opt for the generic one. The ED generic pills are as effective as the branded one if not more. So, opt for the ED generic pills and get rid off this problem for ever. enhancement manhattan penis surgeon penis enlagement surgeries does vigrx work herbal penis enlargment pills free penile enlargment technique safe penis enlargement enlagement manhattan penis surgeon does pnis enlargement work

penis elargement photo

Vitamin B1 also called as thiamine is one of the eight vitamins that make up the powerful group called as the Vitamin B complex. Like all of the B vitamins this nutrient plays a great role in the good health of the body as well as a sound mental health. Proper care and caution should always be considered when it comes to taking in these nutrients especially when it comes to dosages as some may tend to overdose which is never really a good thing and is actually quite as bad as not having any at all. Vitamin B1 serves many purposes in the body. It is an essential part of converting carbohydrates to energy and necessary to the proper functioning of the nervous system, the heart and the musculature system of the body. Thiamine is very important to the brain particularly in terms of emotional health and well being as well as being useful for focus and concentration. There are a variety of physical conditions and diseases associated with deficiencies in Vitamin B1. Symptoms include pain, numbness and tingling in the extremities, muscle weakness and a lack of physical coordination particularly in the larger muscle masses that make up the leg muscles. A deficiency in thiamine can cause enlargement of the heart which can itself lead to congestive heart failure and lung congestion. A severe deficiency in Vitamin B1 can lead to nerve damage, brain damage and even death. Mental symptoms associated with a serious lack of Vitamin B include fatigue, psychosis and confusion. These are a few serious diseases specifically associated with a deficiency of Vitamin B1 including the notorious beriberi disease. There are also two specific types of brain damage associated with severely low levels of Vitamin B1. One is the Korsakoff syndrome which results in confusion and short term memory loss. Another is the Wernicke’s Disease which has symptoms that include vision disturbances and irregularities, an unsteady walk and mental confusion. Despite the obvious importance of Vitamin B1 to many of the body’s important functions a great deal of people regularly fail to meet the recommended daily intake levels of this vitamin. Alcoholics are particularly vulnerable to being deficient in Vitamin B1 because alcohol ruins the B vitamins. Illnesses that force bodily fluids out from the body such as chronic diarrhea can deplete the body’s levels of thiamine because it is a water-soluble vitamin and the body needs to have its steady supply renewed every day. There are also rare conditions that interfere with the body’s ability to use Vitamin B1 properly. Dietary supplements are an affordable, safe and effective way to consistently meet the recommended daily intake levels of Vitamin B1. It is especially important however, when taking supplements to use them with care and attention because taking too much Vitamin B1 can cause adverse side effects and even serious health problems. As with all substances there is such a thing as too much of a good thing. Moderation is always key. While there are certain health situations that do call for higher than average doses of this powerful B vitamin high dosages should only be taken while under the direct supervision of a qualified health care professional. Like any other medication or dietary supplement Vitamin B1 supplements should always be kept well out of the reach of children. ------------------------- Note: This article may be freely reproduced as long as the AUTHOR'S resource box at the bottom of this article is included and and all links must be Active/Linkable with no syntax changes. ------------------------- where to buy vig rx penis enlargment review guide to penile enlargement herbal penile enlargment pills vimax home penis enlargement penis enlarement before and after photo natural pnis enlargement exercise free penis elargement exercise penis elargement photo

To have sex is a natural instinct. Right from the moment an individual reaches puberty his or her view on sex goes on transformation. There comes a moment in life when one is not able to have sex as he or she is aging. For youthfulness one has to have young mind. Mind is the centre of all things. And there is no better medicine to rejuvenate an old individual’s mind than having sex. But to have sex both partners must be psychologically and physiologically be ready. But if the male partner has erectile dysfunction (ED) problem then it is useless even to think about having sex. ED is a commonly found in aged males. It renders his penis useless while having sex. ED can be even found in other non aged males due to several reasons. So, taking of a drug which can get him out of ED is the best option he can choose. Levitra is such an erectile dysfunction treatment pill. Its constituent is Vardenafil Hydrochloride. It is marketed by Bayer Pharmaceuticals Corporation and GlaxoSmithKline. It works by relaxing the penile muscles which in turn leads to increase in blood flow to penis. As the blood is pumped into the penis, it gets harder and gets an erection. The erection lasts long enough to complete love making. Once the male partner ejaculates the erection should fade away. In case erection does not fade after having sex or the erection stays for more than four hours then the person should consult a doctor. The dose for Levitra is a pill a day or as directed by the doctor. If both the instruction is contradictory then doctor’s direction should be followed. The ED pill too can be used by persons with high cholesterol, type 1 or type 2 diabetes or high blood pressure. It is not for use by women or children below 18 years and persons above 65 years. A person with blood pressure, heart ailments or any other diseases whose medication may contain nitrate is not advised to take the pill. Possible side effects of Levitra which are mild and short-term are chest pain, swelling of limbs or ankles, shortness of breath, dizziness, painful erection and allergic reaction to the drug etc. In case of side effects do not hesitate to consult your doctor. Levitra is an ED treatment prescription drug formulated so as that you enjoy your sexual life. But you must have a sexual arousal for having it. It helps you in getting an erection and maintaining it and not in getting you an arousal. pennis enlargement penis enlagement forum penis elargement technique penis enlarement herb cheapest penile enlargment pills penis enargement pills review penile enlargment penis enlargement pic penis elargement photo

“There should be a different therapy for every patient because every patient is different”- William Dempsey (Abbott laboratories) Treatment of erectile dysfunction, no longer remains an agonizing procedure, earlier ED was treated with injection or through surgery, both of these methods were extremely painful and embarrassing, and worst of all, the success was uncertain. The emergence of Viagra, Levitra, and Cialis in 1998(FDA approved prescription drugs for erectile dysfunction) revolutionized the entire concept of the treatment of erectile dysfunction. These drugs provided millions of ED patients around the world with the opportunity to renew and re-stabilize their sexual life. Levitra, Cialis and Viagra are all PDE type 5 inhibitors, they helps the blood vessels in the penis to relax, resulting in flow of blood which causes erection. They are all taken orally, and their basic adverse effects are somewhat familiar. The worth of erectile dysfunction market has grown drastically, it is estimated that by the end of 2006 its income will be $6 billion. 15% of the total male population, nearly 300 million men around the world suffer from impotence and the numbers are not going to decline. This phenomenal market magnetizes several companies to cash in, its no surprise that numerous companies are rushing in with new and efficient methodology to treat erectile dysfunction. The information about the additional medical qualities of Apomorphine led to the innovation of Uprima. This sublingual tablet was launched by the Abbott laboratories U.K. Uprima is a small tablet which dissolves quickly when placed under the tongue, leading to erection within 20 minutes. What’s new about Uprima? UPRIMA is a dopamine receptor agonist that works in the brain to improve diminished erectile function by enhancing the natural signal to the penis following sexual stimulation, similar to the way men normally have erections. UPRIMA works through the central nervous system, producing a series of events that enhances the ability to achieve and maintain penile erection. It is distinct from other oral therapies, which act by blocking the action of certain enzymes involved in the erectile response. Uprima provides plentiful benefits but the most important of all is that its onset is very fast and its effect is similar to natural erection. Some of the common side effects of Uprima are nausea, dizziness and headache, which were in general mild and momentary in nature. Uprima Doses- Uprima is available in 2 mg and 3 mg doses. Conclusion: The quality and the efficiency of Uprima may force other companies indulged in manufacture of erectile deficiency drugs to follow the same path. penis enhancement video enlargment forum free matter penile size top penis enlargment pills vigrx pill free natural penis enargement easy enlargement free penis surgery way natural pennis enlargement exercise penis enlargment traction device penis elargement photo

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth.