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Coldsores, also called fever blisters and oral herpes, are a global epidemic - or pandemic. Coldsores are the visible symptom of an active herpes virus infestation. More specifically, coldsores are the result of the reproduction process of the herpes virus. The World Health Organization estimates 85% to 91% of the world population currently carries the herpes simplex virus type 1 or 2 (HSV-1 and HSV-2). For all practical purposes, that means just about everybody is infected with the coldsores virus. Recorded history shows that this has been true since about 500 years prior to the Roman Empire. HSV-1 is responsible for about 80% of reoccurring coldsores. The other 20% of coldsores are caused by HSV-2. Of those infected with the herpes virus, 76% will have one or several coldsores within the next 12 months. The other 24% often go a lifetime without experiencing any symptom of coldsores. The herpes virus most of the time is latent, or in hibernation, in the nerve ganglia nearest to the site of your coldsores. In the case of facial coldsores, this would be in an area behind the jawbone, near the brain stem. When the coldsores virus becomes active, they travel up the nerve fibers to the surface where they replicate and create those painful coldsores right on the end of the nerves. Coldsores normally occur on the face, appearing on the edge of the lip, called the vermilion border. The nostril is also a common site for coldsores. What most people don't know, however, is coldsores can appear anywhere from the waist up. For example: fingertip coldsores do occur. They're often a much more painful event because of the constant use of the fingers in our daily routine. Coldsores are extremely contagious. The coldsores virus spreads externally, not internally. Kissing is the primary way coldsores are transmitted to others - especially from adults to children. Most people are infected before they're a dozen years old. The lips, mouth and nose are not protected by skin and are an easy target. Coldsores can also spread to anywhere on the body where the virus can find an opening - like a cut on the finger. Although coldsores are not life threatening, coldsores can cause a lot of grief and damage if spread to the eyes with contaminated fingers. This can cause loss of sight. Also, with oral sex, the coldsores can be spread to the vagina or penis, creating the dreaded genital herpes. Coldsores are contagious from the first itching stage to the disappearance of the final red spot. They are most contagious during the open weeping and crust stages. The crust cracks frequently when you move your mouth, as in smiling. The fluid from these coldsores is absolutely teaming with the coldsores virus. Extreme caution must be taken with active coldsores. Coldsores itch and hurt a lot, so we tend to touch them frequently. Then the virus sheds to our fingers - and is easily transmitted to another location or person. Self-control is imperative. Each time you touch your coldsores, you must wash your hands. Keep little bottles of hand sterilizing soap or baby-wipes on hand. Baby-wipes have a sterilizing ingredient and are particularly handy and useful. You can dab the coldsores with them instead of your fingers. This also speeds healing of coldsores. Coldsores are brought on primarily by physical stress. Keep in mind even mental stress will manifest itself physically. Colds (thus the term coldsores), fever (thus the term fever blisters), pregnancy, injury, and nearly any physical trauma can easily bring the virus out of hibernation and cause coldsores. Fact is, upcoming weddings, according to the mail I get, are one of the biggest causes of coldsores. There are a huge variety of treatments for coldsores. These include over-the-counter medications, prescription anti-viral pills and salves, and many natural remedies. You'll find over-the-counter products are mainly comfort medications. None of them have ever proven to shorten your coldsores. One exception to this - some have antibacterial agents. This prevents secondary bacterial infections. If you do get a bacterial infection, and this is common, it will greatly lengthen the healing times and discomfort of your coldsores. Much of the benefit of over-the-counters is a numbing agent to reduce pain. Some contain oils that help keep the scab softer so it doesn't crack. Prescription medications for coldsores, up to this point, have been dismal failures. There are several brand names but the generic name for the active ingredient is acyclovir. The antiviral salve will take, maybe, one or two days off your 3-week coldsores. The antiviral pills helped reduce the number of coldsores for only 47% of the people tested. This was a very short study so it wasn't very accurate. Please note this medication is not to be taken if you're pregnant or going to be pregnant soon. Also, there is some concern for liver damage. Your best option, in fact your ONLY option for coldsores right now, is the variety of known natural remedies for coldsores. In real life studies, natural remedies have shortened the duration of coldsores by as much as 85%. Among these are honey, DMSO, tea tree oil, zinc, lysine, aloe vera, certain herbs and a few others in certain forms and combinations. Coldsores can literally be a real pain. 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Penis enlargement is a special issue in today’s world. Buried underneath tons of unsolicited emails promising the most unlikely results, plagued by dishonest practitioners and obscured by myths and hearsay, the honest traders of this industry have pushed forward with their products and services. Most of the time, men prefer to turn away and say enlargement does not work, even though they have no idea whether this is true or not. Hearsay is just as good as sound, hands-on information if one is not really interested in the issue or if one is afraid of the truth. Many people are keen to dismiss penis exercises as myths even though they are not familiar with the facts. Penis exercises have been around in one form or other for a very long time. Primitive tribes are still using weights, various objects and exercises to force parts of the human body to change size and achieve a new look. The women of the Paduang tribe use metal rings to lengthen their necks, while the people from other tribes hang weights from their lips or ear lobes in order to reach their own standards of beauty. Chinese women of high birth had their feet shrunk in order to fit the local ideal of a sexy look. With all these going on, why should it be so hard to believe that the penis was ignored? Especially since we know that it was not. Various penis enlargement techniques have also been reported, especially among the nomad Arabic tribes. Body enhancement techniques performed by males were always tied to the position of the person in question within the tribe or with the manhood initiation rites. It seems that men found early on that the human body can be modified using devices or exercises. The only traction devices at their disposal for a long period of time were weights, but stretching the penis using one’s own hands was just as good as any device. The basic principle behind body enhancement is the adaptability of the human body in response to external stimuli. Everybody knows that the extra physical effort put into working out at the gym will trigger an increase in the size of the muscles that have to sustain the effort. Thus, repeated exercises focused on the penis, like the ones offered by Penis Health, will force the body to start multiplying the cells that make up the penis tissues and to increase both the length and girth of the penis in order to cope with the new situation. The best known penis enlargement exercise is the Jelq. This exercise is designed to enlarge the penis using milking movements in order to increase the blood flow into the corpora cavernosa, the sponge-like tissues of the penis. The increased blood flow will, in time, force the tissues to expand and increase both the flaccid and erect sizes of the penis. Dr. Brian Richards has conducted a study of penis enlargement exercises in the 1970s and found that jelq helped nearly 90 percent of patients increase their penis size. The gains ranged in size, of course, but it was proven that men could add an inch or even more to their penises. Penis Health is one of the best penis exercises programs around. Hundreds of customers have successfully enlarged their penises using the exercises developed by the people at Penis Health. You don’t have to take anything on faith. Just browse the Mens Network forum and discuss gains with the registered users or browse the Penis Health site and read through the signed testimonials and opinions culled from forum posts. Customers are content with the program and excited about the results. We think there’s nothing better for a man’s confidence than an extra inch or two on his penis. Despite the rabid skepticism of those cannot be bothered to check the facts, common sense and evidence point to the fact that penis enlargement exercises do work. No man who could use an extra inch or two in length or girth should write them off until he’s actually tried them. Many skeptics have been pleasantly surprised by our program of exercises, so why not give it a go? There’s nothing to lose and a whole world of sexual pleasure and self-respect to gain. best penis enargement prosolution pnis enlargement pills where to buy vig rx home penis enargement penis elargement technique permanent penile enlargment medical penile enlargement top penis enlargment pills buy vigrx

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. 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